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Acute cerebral hemorrhage in patients with upper gastrointestinal bleeding study of changes in pituitary hormone
8 blood 5ml,
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levels. 1.3 Statistical Methods SPSS11.0 software, data (x.4-s) that the test used to compare groups. To P 0.05). In Table 1. Table I compared three groups of changes in pituitary hormone

H (mU / L)

'I ~ I (pmol / L) h group (24) 6.68 ± 032 (12) 3843 ± 485B group (26) l492 ± 0.46 ( 10) 6828 ± I ∞ C group (10) 1527 ± 067 (27) 6935 ± 07l Note: with the C group,
moncler jakke dame, p <001; and B-dimensional comparison, △ p <001; the number of cases in brackets 3 to discuss 1019 · input to reach the hypothalamus nerve cells can trigger the release of neuroendocrine factors, promote the secretion of pituitary cells to secrete a variety of target gland hormones to promote, control and regulate the metabolism of target gland hormones.

ute cerebral hemorrhage, intracranial pressure, brain pressure caused by the limitations of hematoma or midline shift of people breaking the ventricular system, under the bottom of the third ventricle of the nucleus is stimulated, the pituitary endocrine disorder, increased pituitary


release, so a lot of the adrenal cortex corticotropin-releasing hormone l2_2, causing an increase in gastric acid secretion, mucus secretion and degeneration, mucosal cells, DNA synthesis decreased, reducing the mucosal barrier, leading to infiltration of hydrogen ions back to the mucosal barrier damage, resulting in mucosal erosion bleeding.

renocorticotropic hormone can promote the secretion of gastric acid and pepsin, increased mucosal barrier damage, major clinical manifestations of ulcer or hemorrhagic gastritis [3] 3. Upper gastrointestinal hemorrhage cerebral hemorrhage mortality up to 50% E. Our data show that cerebral hemorrhage in patients with upper gastrointestinal bleeding, blood


levels can be significantly increased, indicating that patients may be an important factor in upper gastrointestinal bleeding, the blood of patients with cerebral hemorrhage measured


levels in patients with cerebral hemorrhage can be assessed on risk of gastrointestinal bleeding, for early intervention, proton pump inhibitors, H, receptor antagonists application, can reduce gastric acid secretion, gastric mucosal protection, effective prevention and treatment of upper gastrointestinal bleeding 5. Early nasogastric feeding tube, can neutralize stomach acid, protect the gastric mucosa, while additional energy to ensure a stable environment for the body. Treatment of the primary disease, dehydration reducing the intracranial pressure, control blood pressure, reducing the mortality of acute cerebral hemorrhage has a positive meaning. Cerebral hemorrhage in patients with upper gastrointestinal bleeding blood FSH, 'rSH low level mechanism is still not clear, suggesting that the body is facing the critical state of cerebral hemorrhage, and reproduction, metabolism and other functions related to pituitary-gonadal axis, pituitary-thyroid axis being in the inhibitory state, to ensure that all of the potential to mobilize a major blow against the disease.